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NameEmailPhD ProgramResearch InterestPublications
Darwitz, Ben

EMAIL

PHD PROGRAM

RESEARCH INTEREST
Bacteriology, Immunology, Pathogenesis & Infection

Drage, Evan

EMAIL

PHD PROGRAM

RESEARCH INTEREST
Bacteriology, Evolutionary Biology, Molecular Biology

Jackson, Jahnelle

EMAIL

PHD PROGRAM

RESEARCH INTEREST
Bacteriology, Pathogenesis & Infection, Translational Medicine

Kent, Leslie

EMAIL

PHD PROGRAM

RESEARCH INTEREST
Bacteriology, Bioinformatics, Pathology

Shouse, Eva Mei

EMAIL

PHD PROGRAM

RESEARCH INTEREST
Bacteriology, Pathogenesis & Infection, Plant Biology

Thomas, Cynthia

EMAIL

PHD PROGRAM

RESEARCH INTEREST
Bacteriology, Genetics, Pathogenesis & Infection

Azcarate-Peril, M. Andrea
WEBSITE
EMAIL
PUBLICATIONS

PHD PROGRAM
Nutrition

RESEARCH INTEREST
Bacteriology, Bioinformatics, Genomics, Molecular Biology, Systems Biology

We are interested in determining the mechanisms involved in the beneficial modulation of the gut microbiota by prebiotics (functional foods that stimulate growth of gut native beneficial bacteria) and probiotics (live bacteria that benefit their host). Specifically, we aim to develop prebiotic and probiotic interventions as alternatives to traditional treatments for microbiota-health related conditions, and to advance microbiota-based health surveillance methods.

Goldman, William
WEBSITE
EMAIL
PUBLICATIONS

PHD PROGRAM
Microbiology & Immunology

RESEARCH INTEREST
Bacteriology, Biochemistry, Genetics, Immunology, Pathogenesis & Infection

Successful respiratory pathogens must be able to respond swiftly to a wide array of sophisticated defense mechanisms in the mammalian lung.  In histoplasmosis, macrophages — a first line of defense in the lower respiratory tract — are effectively parasitized by Histoplasma capsulatum.  We are studying this process by focusing on virulence factors produced as this “dimorphic” fungus undergoes a temperature-triggered conversion from a saprophytic mold form to a parasitic yeast form.  Yersinia pestis also displays two temperature-regulated lifestyles, depending on whether it is colonizing a flea or mammalian host.  Inhalation by humans leads to a rapid and overwhelming disease, and we are trying to understand the development of pneumonic plague by studying genes that are activated during the stages of pulmonary colonization.

Duncan, Alex
WEBSITE
EMAIL
PUBLICATIONS

PHD PROGRAM
Pharmacology

RESEARCH INTEREST
Bacteriology, Biochemistry, Cell Signaling, Immunology, Pathogenesis & Infection

My lab studies a recently identified pathogen-sensing signaling complex known as the inflammasome. The inflammasome is responsible for the proteolytic maturation of some cytokines and induces a novel necrotic cell death program. We have found that critical virulence factors from certain pathogens are able to activate NLRP3-mediated signaling, suggesting these pathogens may exploit this host signaling system in order to promote infections.  Our lab has active research projects in several areas relating to inflammasome signaling ranging from understanding basic molecular mechanisms of the pathway to studying the role of the system in animal models of infectious diseases.

de Silva, Aravinda
WEBSITE
EMAIL
PUBLICATIONS

PHD PROGRAM
Microbiology & Immunology

RESEARCH INTEREST
Bacteriology, Cell Biology, Immunology, Molecular Biology, Pathogenesis & Infection

We study Borrelia burgdorferi (the agent of Lyme disease) as a model for understanding arthropod vector-borne disease transmission. We also study the epidemiology and pathogenesis of dengue viruses associated with hemorrhagic disease.